Kemerovo, Kemerovo, Russian Federation
In the course of the work, it was found that a large number of genetic features affect the development of atherosclerosis (gene SNPs, telomere length, epigenetic factors, circadian rhythms). Studies of SNP genes, in particular those affecting lipid metabolism, predominate. Relatively new is the study of the relationship between chrononutrition and the development of metabolic diseases.
atherosclerosis, nutritional genetics, nutrigenetics, oligonucleotide polymorphisms, telomere length, epigenetics, chrononutrition
1. Targeting Early Atherosclerosis: A Focus on Oxidative Stress and Inflammation / P. Marchio, S. Guerra-Ojeda, J.M. Vila et al. // Oxidative medicine and cellular longevity, 2019. 8563845
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3. Obesity Programs of nutrition, Education, Research and Assessment (OPERA) Group (2020). Nutrigenetics-personalized nutrition in obesity and cardiovascular diseases / L. Barrea, G. Annunziata, L. Bordoni et al. // International journal of obesity supplements, 2020. 10(1), P. 1–13.
4. Direct-to-Consumer Nutrigenetics Testing: An Overview / M. Floris, A. Cano, L. Porru et al. // Nutrients, 2020. 12(2), P. 566.
5. Metabolic and Vascular Effect of the Mediterranean Diet / A. Tuttolomondo, I. Simonetta, M. Daidone et al. // International journal of molecular sciences, 2019. 20(19), 4716.
6. Polimorfizm genov belkov – regulyatorov vospaleniya pri ateroskleroze, oslozhnennom razvitiem ostrogo infarkta miokarda / V.I. Konenkov, A.V. Shevchenko, V.F. Prokof'ev, M.I. Voevoda // Ateroskleroz, 2011. T. 7, № 1, S. 5–18.
7. Hotina V.A., Suhorukov V.N., Kashirskih D.A., Sobenin I.A., Orehov A.N. Metabolizm holesterina v makrofagah. Kompleksnye problemy serdechno-sosudistyh zabolevaniy. 2020;9(2):91-101.
8. Pat. 2698092, Sposob vyyavleniya predraspolozhennosti k zabolevaniyu aterosklerozom na osnove opredeleniya ekspressii genov, vovlechennyh v nakoplenie holesterina / patentoobladatel' FGBNU «NII obschey patologii i patofiziologii». – zayav. 2018112967; opubl. 10.04.2018.
9. Integrative studies implicate matrix metalloproteinase-12 as a culprit gene for large-artery atherosclerotic stroke / H. Mahdessian, L. Perisic Matic, M. Lengquist, et al. // Journal of internal medicine, 2017. 282(5), P. 429–444.
10. Rol' genov antioksidantnoy sistemy v formirovanii klinicheskih fenotipov ishemicheskoy bolezni serdca / A.A. Podol'skaya, A.S. Galyavich, E.V. Maykova i dr. // Kazanskiy medicinskiy zhurnal, 2013. T. 94, № 2, S. 228–234.
11. Abdullah, M.M. Nutrigenetics of cholesterol metabolism: observational and dietary intervention studies in the postgenomic era / M.M. Abdullah, P. J. Jones, P.K. Eck // Nutrition reviews, 2015. 73(8), 523–543.
12. Tackling Atherosclerosis via Selected Nutrition / A. D. Vesnina, A. Yu. Prosekov, V. V. Atuchin [et al.] // International Journal of Molecular Sciences, 2022. Vol. 23, No 15, P. 8233.
13. Genes and eating preferences, their roles in personalized nutrition / A. Vesnina, O. Kozlova, A. Prosekov, V. Atuchin // Genes, 2020. Vol. 11, No 4, P. 357.
14. Association between diet quality indexes and the risk of short telomeres in an elderly population of the SUN project / A. Ojeda-Rodríguez, I. Zazpe, L. Alonso-Pedrero et al. // Clinical nutrition, 2020. 39(8), 2487–2494.
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16. Fishbein, A. B. Circadian disruption and human health / A.B. Fishbein, K.L. Knutson, P.C. Zee // The Journal of clinical investigation, 2021. 131 (19), e148286.
17. Possible contribution of chronobiology to cardiovascular health / M. Sato, T. Matsuo, H. Atmore et al. // Frontiers in physiology, 2014. 4, 409.
